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Oxidation: Publications and Research from SwissMixIt

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Fatty acid B oxidation is a major source of energy in the mitochondria; ultimately generating the reducing agents flavin adenine dinucleotide (FADH2) and nicotinamide adenine dinucleotide (NADH+) to serve as electron donors to the respiratory chain for oxidative phosphorylation and ATP generation. Lipids are the substrate largely responsible for energy supply during submaximal exercise. Lipids as a fuel source for energy supply during submaximal exercise originate from subcutaneous adipose tissue derived fatty acids (FA), intramuscular triacylglycerides (IMTG), cholesterol and dietary fat.

Keywords: Fatty acid oxidation disorders (FAODs), mitochondrial β-oxidation, hypoketotic hypoglycemia, carnitine, trifunctional protein, Fat oxidation, Substrate oxidation, Dietary fat oxidation, Crossover concept, Maximal fat oxidation, PDH activity, Fat adaptation, Ketogenic diet, Cpt-1, Carnitine, oxidative deterioration, hydroperoxide, volatile compounds, aldehydes, thiobarbituric acid reactive substances (TBARs), analytical methods, free radicals, Aging, Antioxidants, Phenolic compounds, Skin aging, pro-oxidant, reducing power, cyclic voltammetry, oxidative damage markers

Summary of Abstracts:

Fatty acid β-oxidation is a major source of energy in the mitochondria; ultimately generating the reducing agents flavin adenine dinucleotide (FADH2) and nicotinamide adenine dinucleotide (NADH+) to serve as electron donors to the respiratory chain for oxidative phosphorylation and ATP generation. Lipids are the substrate largely responsible for energy supply during submaximal exercise. Lipids as a fuel source for energy supply during submaximal exercise originate from subcutaneous adipose tissue derived fatty acids (FA), intramuscular triacylglycerides (IMTG), cholesterol and dietary fat. These sources of fat contribute to fatty acid oxidation (FAox) in various ways. The regulation and utilization of FAs in a maximal capacity occur primarily at exercise intensities between 45 and 65 percent VO2max, is known as maximal fat oxidation (MFO), and is measured in g/min. Fatty acid oxidation occurs during submaximal exercise intensities, but is also complimentary to carbohydrate oxidation (CHOox). Due to limitations within FA transport across the cell and mitochondrial membranes, FAox is limited at higher exercise intensities. The point at which FAox reaches maximum and begins to decline is referred to as the crossover point. Exercise intensities that exceed the crossover point (65 Percent VO2max) utilize CHO as the predominant fuel source for energy supply.

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Email: greg@swissmixit.com


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